@Article{IPB-2195, author = {Ibañez, C. and Poeschl, Y. and Peterson, T. and Bellstädt, J. and Denk, K. and Gogol-Döring, A. and Quint, M. and Delker, C.}, title = {{Ambient temperature and genotype differentially affect developmental and phenotypic plasticity in Arabidopsis thaliana}}, year = {2017}, pages = {114}, journal = {BMC Plant Biol}, doi = {10.1186/s12870-017-1068-5}, url = {https://dx.doi.org/10.1186/s12870-017-1068-5}, volume = {17}, abstract = {BackgroundGlobal increase in ambient temperatures constitute a significant challenge to wild and cultivated plant species. Forward genetic analyses of individual temperature-responsive traits have resulted in the identification of several signaling and response components. However, a comprehensive knowledge about temperature sensitivity of different developmental stages and the contribution of natural variation is still scarce and fragmented at best.ResultsHere, we systematically analyze thermomorphogenesis throughout a complete life cycle in ten natural Arabidopsis thaliana accessions grown under long day conditions in four different temperatures ranging from 16 to 28 °C. We used Q10, GxE, phenotypic divergence and correlation analyses to assess temperature sensitivity and genotype effects of more than 30 morphometric and developmental traits representing five phenotype classes. We found that genotype and temperature differentially affected plant growth and development with variing strengths. Furthermore, overall correlations among phenotypic temperature responses was relatively low which seems to be caused by differential capacities for temperature adaptations of individual accessions.ConclusionGenotype-specific temperature responses may be attractive targets for future forward genetic approaches and accession-specific thermomorphogenesis maps may aid the assessment of functional relevance of known and novel regulatory components.} } @Article{IPB-1445, author = {Costa, C.T. and Strieder, M.L. and Abel, S. and Delatorre, C.A.}, title = {{Phosphorus and nitrogen interaction: loss of QC identity in response to P or N limitation is anticipated in the \textit{pdr23} mutant}}, year = {2011}, pages = {219-229}, journal = {Braz J Plant Physiol }, url = {http://www.scielo.br/scielo.php?script=sci_arttext&pid=S1677-04202011000300006&lng=en&nrm=iso}, volume = {23(3)}, abstract = {Changes in root architecture are an important adaptive strategy used by plants in response to limited nutrient availability to increase the odds of acquiring them. The quiescent center (QC) plays an important role by altering the meristem activity causing differentiation and therefore, inducing a determinate growth program. The arabidopsis mutant pdr23 presents primary short root in the presence of nitrate and is inefficient in the use of nucleic acids as a source of phosphorus. In this study the effect of the pdr23 mutation on the QC maintenance under low phosphorus (P) and/or nitrogen is evaluated. QC identity is maintained in wild-type in the absence of nitrate and/or phosphate if nucleic acids can be used as an alternative source of these nutrients, but not in pdr23. The mutant is not able to use nucleic acids efficiently for substitute Pi, determinate growth is observed, similar to wild-type in the total absence of P. In the absence of N pdr23 loses the expression of QC identity marker earlier than wild-type, indicating that not only the response to P is altered, but also to N. The data suggest that the mutation affects a gene involved either in the crosstalk between these nutrients or in a pathway shared by both nutrients limitation response. Moreover loss of QC identity is also observed in wild-type in the absence of N at longer limitation. Less drastic symptoms are observed in lateral roots of both genotypes. } } @Article{IPB-831, author = {Schilling, S. and Stenzel, I. and von Bohlen, A. and Wermann, M. and Schulz, K. and Demuth, H.-U. and Wasternack, C.}, title = {{Isolation and characterization of the glutaminyl cyclases from \textit{Solanum tuberosum} and \textit{Arabidopsis thaliana}: implications for physiological functions}}, year = {2007}, pages = {145-153}, journal = {Biol. Chem}, volume = {388}, }